Lung cancer results from effects of smoking along with multiple genetic components. A new study identifies two main pathways for the role of chromosome 15q25.1 -- a leader in increasing susceptibility to lung cancer -- in modifying disease risk. One pathway is implicated in nicotine dependence. The other plays a part in biological processes such as nutrient transfer and immune system function. The findings increase our understanding of lung cancer cause and development.
Original Article: Insight into development of lung cancer